Inclusion body myositis (IBM): clinical features
Written and reviewed by Dr Ibrahim Imam, Consultant Neurologist. Last reviewed .
Inclusion body myositis (IBM): clinical features. IBM is a late-onset inflammatory myopathy. Fully referenced neurology checklist.
Background
- IBM is a late-onset inflammatory myopathy
- It is typically acquired
- It may however be hereditary
- It has both inflammatory and degenerative componentsÂ
Demographic features
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Classical weakness pattern
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Other weakness features
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Other features
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Associated disorders
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References
- Amato AA, Russell JA. Neuromuscular Disorders. McGraw Hill New York 2008 pp681-719.
- Solorzano GE, Phillips LH. Inclusion body myositis: diagnosis, pathogenesis, and treatment options. Rheum Dis Clin N Am 2011; 37:173-183.
- Dalakas MC. Sporadic inclusion body myositis-diagnosis, pathogenesis and therapeutic strategies. Nature Clin Pract Neurol 2006; 2:437-447.
- Dabby R, Lange DJ, Trojaborg W, et al. Inclusion body myositis mimicking motor neuron disease. Arch Neurol 2001; 58:1253-1256.
- Damian L, Login CC, Solomon C, et al. Inclusion body myositis and neoplasia: a narrative review. Int J Mol Sci 2022; 23:7358.
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